Sinciput Sanguisugae
Published on Nov 18, 2015
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PRESENTATION OUTLINE
My critter is called "Sinciput Sanguisugae", which means "head leech" in Latin.
Photo by kevin dooley
It is a parasitic organism that is closely related to the bacteria T. gondii and it's ancestors are other other floridean starch accumulating and glycogen storing organisms.
Photo by Werner Kunz
It inhabits cities and thrives in unkept and/or unclean places. (Garbage, fecal matter, sewers)
Photo by MagnusL3D
It travels to the brain through the Endocrine system and infects the host inside of the cranium.
Photo by kevin dooley
Adaptations
- Utilizes molecular mimicry
- Enters the body through nose and tear ducts
- Two neurological hubs for sustainability
- White tendrils leech sugars and oxygen from host
- Only identification is eye color
Photo by VinothChandar
It also manifests itself inside of the cranium. It evolved this trait over time.
Photo by Alyssa L. Miller
Within the Sinciput Sanguisugae population, there was variation. Some utilized the stomach to survive, some utilized the cranium.
Photo by Robert S. Donovan
When antibiotics rose to prominence, a struggle for survival began.
Photo by somegeekintn
Having more control over the production of various substances inside of the body, the brain favoring variation had a higher rate of survival and reproduction.
Apoptosis is the process of programmed cell death.
(PS) exposure on the cell surface indicates apoptosis, but has also been related to evasion mechanisms of parasites, a concept known as apoptotic mimicry.
Toxoplasma gondii mimics apoptotic cells by exposing PS, inducing secretion of TGF-beta1 by infected activated macrophages leading to degradation of inducible nitric oxide (NO) synthase, NO production inhibition and consequently persisting in these cells.
The parasite utilizes this cell mimicry to avoid detection upon initial infection.
(PS) exposure on the cell surface indicates apoptosis, but has also been related to evasion mechanisms of parasites, a concept known as apoptotic mimicry.
Toxoplasma gondii mimics apoptotic cells by exposing PS, inducing secretion of TGF-beta1 by infected activated macrophages leading to degradation of inducible nitric oxide (NO) synthase, NO production inhibition and consequently persisting in these cells.
The parasite utilizes this cell mimicry to avoid detection upon initial infection.
Photo by Leo Reynolds
Having survived and reproduced, they will pass this trait on to the next generation. Over time, they could evolve specialized functions for manipulating the brain.
Photo by i k o
